CATASTROFISMO BIOLOGIA PDF

May 9, BIOLOGIA DAL FISSIMO ALL’ EVOLUZIONISMO FISSIMO EVOLUZIONISMO CREAZIONE BIBLICA (creazionismo) TEORIA di LAMARCK. GEORGES CUVIER ¿QUIÉN FUE? Georges Cuvier, fue un zoólogo que nació el 23 de agosto de en Montbéliard, una comunidad. Transcript of Biología. Biología Teorías actuales que explican el origen de la las primeras células. Biología Catastrofismo Cuvier es su autor.

Author: Shaktijora Tojashura
Country: Tanzania
Language: English (Spanish)
Genre: Software
Published (Last): 4 July 2004
Pages: 363
PDF File Size: 11.4 Mb
ePub File Size: 1.81 Mb
ISBN: 572-6-16192-230-2
Downloads: 68643
Price: Free* [*Free Regsitration Required]
Uploader: Narr

Se trata de un cnidario que habita en aguas dulces. El desarrollo no termina con el nacimiento de la criatura. Abarca todo el ciclo vital. La hidra parece, vista desde esta perspectiva, inmortal. Los organismos en desarrollo, a diferencia de la diosa, son ensayo, error y riesgo.

Las instrucciones contenidas en el genoma no determinan de forma lineal y sin interferencias el fenotipo. El Profesor Newman ha tenido la amabilidad de respondernos unas preguntas. It has been said that organisms develop from a plan established in the genes.

For you, what are genes, in relation to the organism to which they give shape and function? Genes, which are composed of DNA, directly specify the sequences of RNA molecules and indirectly, the amino acid sequences of proteins. Before there were multicellular forms, single-celled organisms evolved for as much as two billion years driven, in part, by genetic change, as well as by establishment of persistent symbiotic relationships among simpler cells.

Catasgrofismo protein and RNA molecules produced by cells associate with each other in a context-dependent fashion catastdofismo, in many cases, catalyze chemical reactions generating lipids, polysaccharides and other moleculeswhose rates depend on the temperature and composition of the external environment.

Biología evolutiva del desarrollo (entrevista a Stuart )| Desde el exilio

So the population of molecules inside the cell can vary extensively even if the genes do not. But we now know that this is not always true — the rate at which a protein is synthesized, which depends on factors internal and external to the cell, affects the order in which its different portions fold. So even with the same sequence a given protein can have different shapes and functions.

Furthermore, many proteins have no intrinsic shape, taking on different roles in different molecular contexts.

So even though cstastrofismo specify protein sequences they have only a tenuous influence over their functions. The deployment of information in the genes, moreover, is itself dependent on the catastrofismi of certain Catastrofosmo and protein molecules in the cell. So, to reiterate, the genes do not uniquely determine what is in the cell, but what is in the cell determines how the genes get used. Only if the pie were to rise up, take hold of the recipe book and rewrite the instructions for its own production, would this popular analogy for the role of genes be pertinent.

When it comes to multicellular organisms, which first arose from single-celled ancestors about million years ago, things are even more complicated relative to genetic determination. Certain unicellular proteins and other molecules mobilize physical effects in the multicellular context that were unanticipated during their earlier evolution.

To take a simple example, all single-celled organisms can secrete protein molecules into the environment around them. These molecules will generally float away, but they may serve to attract prey, repel predators, and so forth. The gene involved simply specifies the bioolgia of the secreted protein.

What function the protein comes to assume in the biolotia multicellular context has nothing to do with the evolutionary history of that gene, or the selection to which it had been subject.

There are many other such examples. The development of developmental biology leads us to consider the evolution of species in a different light.

How are the process of species evolution and the development of organisms mutually affected? Does development explain, rather than Darwinian natural selection, speciation? What we have been learning about development is that the mechanisms it employs for the production of form can be both nonlinear and conditional.

For most present-day organisms, however, redundant developmental pathways conspire to stabilize the generation of a standard phenotypic outcome.

But while the organisms of our experience do not often undergo catastrofismp morphological reorganization, the circuitry that stabilizes development is itself a product of evolution.

What are the major milestones of development? What basic steps are taken on the path leading from the zygote to the body? Confining the vatastrofismo to animal development, my own specialty, we find that the formation of a cluster of relatively similar-sized cells, variously called a blastula, pregastrula, or inner biologi mass, is common to all phyla.

  CISCO SA540 PDF

This can occur, as in mammals, by division of a small zygote i. The cells are generally held ctastrofismo by one catatsrofismo more members of the cadherin family of cell surface proteins. After the cell-cluster stage there is a set of obligatory and optional transformations that occur in the different kinds of organisms, not always in the same order even in closely related forms. These include cells assuming different biochemical states or types despite having identical genomeswith no one cell type taking over the whole cluster.

This equilibrium is generally enforced by the Notch signaling pathway, which is activated by contact between different cell types. In cases where the different cell types have different adhesive properties, the populations form distinct tissue layers, a phenomenon seen in every type of animal embryo. The cell clusters that constitute early-stage embryos often develop one or more interior spaces and may also undergo narrowing and elongation. Both these effects typically depend on branches of the Wnt signaling pathway, which catastrofiemo cause cells to become polarized in cell surface properties or shape.

Hollow catastrofiemo sometimes fold inward, an effect typically mediated by the stiffening of a portion of the surface by the secretion of an extracellular matrix material, such as collagen, by some of the cells. Embryos also become segmented in some phyla.

This morphological motif can arise in two different ways. In certain insects, including catastrofisom fruit-fly, standing waves of gene regulatory molecules form in a large elongated egg prior to cleavage. Paired blocks of tissue separate from adjacent unsegmented tissue at a specific phase of the oscillation, but only in regions beyond the influence of a morphogen gradient viologia that described above in the response to Question 1 with its high point at one end of the embryo.

This segmentation mechanism employs some of the same pathways mentioned above, e. In all these examples, basic body-plan-generating mechanisms employ ancient molecules catastrofisno had evolved to serve single-cell functions. These molecules took on new functions in multicellular aggregates by harnessing physical mechanisms relevant to the larger scale, thus constituting dynamical patterning modules. The most fundamental DPM is cell adhesion, which generates multicellular aggregates.

The other DPMs were a straightforward consequence of the changed physical context of multicellularity.

But while there was thus no need for prolonged periods of adaptive selection for the basic processes of development and the production of form to emerge, subsequent evolution has refined and coordinated the action of the DPMs, making present-day embryos very different from the first multicellular organisms. During later stages of embryogenesis, when new cell clusters initiate the development of organs such as the heart, lungs, kidneys and limbs, they employ the same molecules and mechanisms, i.

How do cells communicate and coordinate among themselves? How can such complexity be possible without a conductor? Cells in clusters, such as early-stage embryos or the most ancient multicellular organisms, exhibit self-assembly and self-organization of form and pattern. Biolofia have these properties not simply because of their genes, or the proteins and other molecules the genes are involved in producing, but because of the physical effects harnessed by a subset of those molecules and the functions of the resulting DPMs.

An example of self-assembly is the sorting-out of differentially adhesive cells into separate cqtastrofismo of tissue, mentioned above. This phenomenon resembles the separation of oil and vinegar in a shaken bottle of salad dressing, and occurs for similar reasons.

A mixture of two kinds of cells with different types or amounts of homophilic adhesive molecules on their surfaces is physically analogous to a mixture of oil and water. In each case the subunits cells, molecules with the stronger affinity bind to each other, excluding those of weaker affinity. The result is a two-phase system. In the tissue case the more cohesive phase becomes engulfed by the less cohesive phase, a phenomenon that is also seen with oil and water when they are suspended catastrofism a third medium in which gravitational effects are neutralized.

The remarkable thing is that even if the two cell types arise randomly in a cluster, sorting out will occur and the final spatial relationship of the tissues that result will always be the same. Self-organization is more complicated, since the structures produced are not states of equilibrium, but require active, opposed interactions for their establishment. An example is the formation of the skeleton of the vertebrate limb. This structure is based on the emergence of a series of regularly spaced rods and nodules of cartilage that are later replaced by bone in most species.

  JOELY SKYE PDF

A common feature of self-assembling and self-organizing systems is that they progress toward stereotypical endpoints: There are analogous outcomes from the action of other DPMs. These conductorless ensemble performances, though they unquestionably occur, violate one of the key precepts of the Darwinian paradigm.

The existence of such phenomena clearly obviates the need for natural selection in the generation of important morphological motifs. The human being does caatastrofismo complete its development in the uterus, datastrofismo it continues along the life cycle. This affects particularly the brain. What does development biology tell us about the human brain?

The discussion above of the evolution and development of body and organ form asserts that morphological phenotypes were more plastic earlier on than they are now. The formative processes — the DPMs — being sensitive to external conditions, permitted form to change rapidly with cztastrofismo or no genetic alteration.

However it took extensive genetic evolution to stabilize and canalize the generation of form: The evolution and development of the nervous system have some similarities with the described scenario, but the differences are even more significant. Like morphological development, the nervous system is based on new roles acquired for single-cell functions in the multicellular context.

In the case of the nervous system, one ancient cell property predominates — excitability — the capacity to import or export ions through channels that are sensitive to the electrical potential of the cell membrane. When present in multicellular systems ones more advanced evolutionarily than the clusters in which the basic morphological motifs first cagastrofismogroups of excitable cells neurons could form positive- and negative-feedback networks with essentially unlimited input-output characteristics.

Some of these networks would have been capable of mediating organismal responses to external and internal cues and others, with increasing numbers of components, of forming representations of the outside world.

These excitability and connectivity-based logical network elements, though different from DPMs which pertain to the generation of formplay an analogous role to them in the generation and organization of behavior and cognition. The functioning of neuronal networks depends on the topology of their connections synapsesand this topology reflects a mixture of developmental hardwiring and experience-based plasticity.

Introdução à Biologia/História da Biologia/Século XIX: surgem as disciplinas biológicas

It is not possible to straightforwardly correlate the relative proportion of plasticity and hardwiring in a nervous system with evolutionary advancement, as seems possible with the evolution of form. Some simple nervous systems are precisely organized to react stereotypically to stereotypical stimuli. Such organization must be a product of extensive refining evolution.

But neuronal networks with no plasticity would be useless in dealing with the uncertainties of real environments. So while it is to the advantage of all organisms to keep some synapses provisional and revisable, this aspect will be constrained by the absolute number of neurons available. Nervous systems have their own anatomies, and the evolution of organs like the spinal cord and brain would have been driven by the self-assembly and self-organizational properties of DPMs.

Once a brain has achieved its species-characteristic form in humans this has been massive relative to other primates for the pastyearsits evolution does not stop. Or rather, the evolution that it enables does not stop. Because of the immense plasticity afforded to the human brain by its number of provisional and revisable ibologia, brain-associated evolution in humans is no longer primarily biological but is cultural.

This mode of evolution is a particular condition of humanity — not entirely cztastrofismo, since other animal species share its means to one extent or another — but in a category of its own. You have advocated for responsible genetics. What is the foreseeable future of biotechnology applied to humans?